MolTransport :

Transport Type unknown
Owner Public
URN urn:agi-MolTransport:inout-urn:agi-llid:10062:out-urn:agi-cas:50-99-7::unknown
Entities glucose <--- NR1H3
References 4
Connectivity 2
Effect unknown

Tissue blood
Organism Mus musculus
MedLine Reference 12923232:1187
Sentence Because Cao et al. ( 19 ) used peripheral blood for their studies, and it has been recently shown that LXR may upregulate tissue glucose uptake, perhaps the major plasma glucose-lowering effect of LXR ligands results from increased peripheral tissue glucose uptake.
Journal Lipid Res
Journal Reference v44 i11 p2039 (2003)
Journal Link http://www.jlr.org/cgi/content/full/44/11/2039

Organism Mus musculus
MedLine Reference 12697904:8
CellType adipocyte
Sentence Consistent with their effects on GLUT4 expression, LXR agonists promote glucose uptake in 3T3-L1 adipocytes in vitro.
CellLineName 3T3 L1

Organism Mus musculus
MedLine Reference 12138207:1316
CellType adipocyte
Sentence Taken together, these data suggest that activation of LXR increases glucose uptake and storage, at least in part, due to an increase in GLUT1.
Journal Mol. Cell. Biol
Journal Reference v22 i16 p5989 (2002)
Journal Link http://mcb.asm.org/cgi/content/full/22/16/5989
CellLineName 3T3 L1

Tissue fat
Organism Mus musculus
MedLine Reference 12554745:1198
Sentence These authors rather demonstrated new metabolic roles for LXR in adipose tissue with effects on metabolic pathways such as increased basal glucose uptake, glycogen synthesis, cholesterol synthesis, and release of nonesterified fatty acids.
Journal Mol. Endocrin
Journal Reference v17 i2 p172 (2003)
Journal Link http://mend.endojournals.org/cgi/content/full/17/2/172